Knock em’ Out: Tackling Concussions/CTE Head On

January 13, 2017



That’s the estimated number of people in the United States who sustain mild traumatic brain injuries (mTBI, i.e. concussions). But that may be just the tip of the iceberg, as some estimates have the total of sports-related concussions per year up to 3.8 million! The CDC has officially recognized that sports concussions in the U.S. have reached epidemic levels.


And yet, even with these terrifying estimates, the reality may be even worse. While many of the complications from mTBI’s and concussions manifest immediately following injury or soon after, they are often unrecognized, undiagnosed, underreported, and ultimately, untreated. A few of the many potentially debilitating symptoms that can arise as a consequence of repetitive mTBI’s include: chronic headaches, dizziness, difficulty concentrating, reduced attention, chronic fatigue, irritability, depression, and impulsivity.


Since the early 21st century, the scientific community has taken considerable measures examining the impact of such repetitive brain trauma, with the most remarkable outcome developing into what has been termed Chronic Traumatic Encephalopathy or more commonly known as CTE, a neurodegenerative disease[1] whose symptom presentation closely resembles that of Alzheimer’s disease. Moreover, there is substantial support indicating long-term brain damage, specifically with regards to cognitive (i.e. impaired attention and concentration, language difficulties, memory problems, executive dysfunction), behavioral/psychological (i.e. aggression, agitation, apathy, impulsivity, depression, suicidality), and motor symptoms (i.e. poor articulation, impaired balance and coordination, gait disturbances, parkinsonism).


With symptoms arising days, weeks, months and even years following initial time of impact, athletes of high contact sports including football, soccer, hockey, baseball, rugby, mixed martial arts, boxing, lacrosse and wrestling are at an especially heightened risk of developing CTE. In a recent study conducted at Boston University’s CTE center, researchers found evidence of CTE in 90 out of 94 brains or 95% of the NFL athletes post mortem at autopsy[2]. Given such alarming statistics, it is critical to understand the distinct criteria of CTE for the prevention, diagnosis and treatment of those at risk for suffering from the disease.


Picture this scenario…




After decades of wear and tear to your body, you can no longer ignore the day-to-day challenges you face, as they have greatly impacted not just your quality of life, but that of your loved ones as well. After years of unpredictable mood swings, forgetfulness, headaches, emotional outbursts, and sleep disturbance you decide to visit a neurologist for a formal medical evaluation, with the hopes of some answers and treatment. You explain your symptomatology and acknowledge an overwhelming feeling of “loss of control.” As a former professional athlete, you report being knocked unconscious a total of 4 times throughout the duration of your amateur and professional career; however, after further inquiry, you admit to having experienced frequent “concussion-like” episodes without loss of consciousness.


Your spouse reports noticeable changes in your relationship with your family and friends, increased sadness and apathy, isolation, and extreme emotional instability, stating that she no longer recognizes the person you have become. Although the neurologist states that only postmortem biopsies can truly confirm CTE, he reports a high likelihood of the early signs of CTE, and declares that there is currently no known cure for the disease. The optimism that you had of alleviating your symptoms is instantly gone as you leave the office with an overpowering sense of hopelessness and despair.


However, what if you were told that there is a treatment that is not only 100% safe but has been scientifically proven, over and over again, to reduce and even reverse the pathological markers that are associated with CTE. Well, there is…




MOLECULAR HYDROGEN, or what is more commonly termed H2, is set to revolutionize the treatment of anyone suffering from the effects of concussions. As the most effective antioxidant in existence, molecular hydrogen is distinct from other antioxidants and therapies for a number of reasons, including:


  • It acts as a “selective” antioxidant, scavenging only the most toxic free radicals in our body.

  • It is the smallest element in the universe, and therefore it can easily dissolve into and move across ALL cell membranes, the blood brain barrier of the central nervous system, and ultimately ANY compartment of our bodies – NO OTHER antioxidant has the ability to penetrate such biological membranes.

  • It has the power to initiate the body’s own self-defense system against free radicals.

  • It has the ability to prevent the formation of the most toxic free radicals.

  • It has incredibly powerful anti-inflammatory properties, addressing the root cause of all diseases.

  • It is 100% safe with absolutely zero negative side effects. It is virtually impossible to overdose, since any excess H2 is expelled through the lungs.


Ultimately, molecular hydrogen not only alleviates the vast majority if not all of the symptoms, it addresses the root cause of the disease process itself.


In sum, the therapeutic significance of molecular hydrogen is nothing short of a miracle. H2 is the ONLY treatment available that has the potential to prevent the onset of CTE and even reverse the symptoms that have for too long taken away from the simple joys of your daily life. Finally, a simple, safe, and effective therapy that will enable you to live a long, healthy, and happy life alongside your loved ones.



To find out more details about H2 and how you can implement it into your life immediately, click here.



[1] An umbrella term for a range of conditions which primarily affect the neurons (the building blocks of the nervous system (brain and the spinal cord)) in the brain. Common neurodegenerative diseases include Parkinson’s Disease, Alzheimer’s disease, Huntington’s disease, etc.). Neurons do not typically replace themselves so when they become damaged and die off they cannot be replaced by the body.


[2] Bieniek KF, Ross OA, Cormier KA, Walton RL, Soto-Ortolaza A. Johnston AE, et al. (2015). Chronic traumatic encephalopathy pathology in a neurodegenerative disorders brain bank. Acta Neuropathol. 130: 877-889.


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